Jump to main content.

Staten Island/New Jersey Urban Air Toxics Assessment Project

Printer-friendly version

The project was a program of ambient air monitoring and meteorological data collection from October 1987 through September 1989, and indoor air sampling from July 1990 through mid-March 1991. An emission inventory was conducted to assist source identification and support risk management where the risk assessment indicated such a need.

This study examined the levels of toxic air pollutants in the Staten Island/New Jersey area and was designed to assess the health risks (cancer and noncancer) to the residents of Staten Island due to the emission of these toxic air pollutants. Estimation of risk of cancer and noncancer effects from lifetime inhalation exposure to air toxics released from point, area, and mobile sources. A limited study of indoor air and an odor monitoring study was also carried out.

Completed reports available from National Technical Information Service (NTIS), telephone number (703) 878-4650.

None Provided

Show details for General InformationGeneral Information

Show details for Assessment and Analysis MethodsAssessment and Analysis Methods

Show details for Risk Assessment Project DesignRisk Assessment Project Design

Hide details for FindingsFindings
This section describes the results of the assessment, the stakeholders involved, and any future plans related to the assessment.

Printer-friendly version of this section

1. What are the media concentration or exposure results?

Concentrations of the VOCs were rather uniform among the sites. Concentrations were statistically significantly higher at one site for tetrachloroethene, and lower at one site for toluene and o-xylene. The tetrachloroethene sources were two dry cleaners and a pumping station at a publically owned treatment facility.

For chemicals included in the quantitative risk assessment, the annual average concentrations measured in this study were generally in the same concentration ranges as those for other urban areas nationwide. For chromium, nickel, cadmium, and vanadium, concentrations were higher in this study than in other urban areas. This is based on comparison to the EPA Urban Air Toxic Monitoring Program (UATMP) results for 1988 and 1989.

The indoor air study showed that concentrations of 13 VOCs in four houses were generally similar to concentrations in several other databases for indoor air. Concentrations of 8 VOCs, including toluene and benzene, were usually higher indoors than outdoors.

2. What are the individual cancer risk estimate results?

The cumulative cancer risk estimate was 96 or 123 per million, depending on the assumption concerning ambient air concentrations of hexavalent chromium.

2.a. What is the cancer risk estimate for the maximum exposed individual (MEI)? What HAPs drive the risk?

2.b. What is the maximum individual risk (MIR)? What HAPs drive the risk?

3. What are the individual non-cancer risk estimates: MEI, MIR, worker? What HAPs drive the risk?

4. What are the target-organ specific hazard indices (TOSHI)? If they weren't developed, why not?

5. What are the population risk results?

6. What other results were found as part of this project?

The estimated cancer and noncancer toxicity risks associated with benzene were consistently higher than those estimated for the other pollutants. The next highest risk for ambient air exposure were associated with nickel, chromium, arsenic, and tetrachloromethane.
The additive risk assessment yielded a maximum hazard index of 2 for noncancer toxicity; the effects were blood formation problems and respiratory tract irritation.

7. What pollutants were identified as being of concern? On what basis?

The cancer risk drivers are benzene, formaldehyde, chromium, tetrachloromethane, trichloromethane, and cadmium. The noncancer risk drivers are benzene, nickel, chromium, and tetrachloromethane.

8. What sources of pollution were identified as being of concern?

Mobile sources, refineries, and gasoline stations for their contributions to benzene. Dry cleaners and pumping stations of Publically Owned Treatment facility for their contributions to tetrachloroethene.

Back To Top

Show details for Outcomes and Reduction ActivitiesOutcomes and Reduction Activities

Show details for Public InvolvementPublic Involvement

Show details for Completed ReportsCompleted Reports

Jump to main content.